Tumour-induced osteomalacia.
| dc.contributor.author | Fernando, Devaka | |
| dc.contributor.author | Nuam, Cing San | |
| dc.contributor.author | Tun, Thein Zaw | |
| dc.date.accessioned | 2022-11-01T14:00:56Z | |
| dc.date.available | 2022-11-01T14:00:56Z | |
| dc.date.issued | 2022-07 | |
| dc.identifier.uri | http://hdl.handle.net/20.500.12904/15925 | |
| dc.description.abstract | Phosphate is important for normal mineralisation of bone. Phosphate is important in its own right for neuromuscular function, and profound hypophosphataemia can be accompanied by encephalopathy, muscle weakness and cardiomyopathy. Hypophosphataemia can be due to intracellular uptake of phosphate from the extracellular fluid, reduced intestinal phosphate absorption, increased renal excretion, decreased renal tubular absorptive capacity and genetic defects in renal tubule phosphate transporters. | |
| dc.description.uri | https://www.rcpjournals.org/content/clinmedicine/22/Suppl_4/24 | en_US |
| dc.publisher | Clinical Medicine Journal | en_US |
| dc.subject | Phosphate | en_US |
| dc.subject | Hypophosphataemia | en_US |
| dc.subject | Muscle weakness | en_US |
| dc.subject | Encephalopathy | en_US |
| dc.title | Tumour-induced osteomalacia. | en_US |
| dc.type | Article | en_US |
| rioxxterms.funder | Default funder | en_US |
| rioxxterms.identifier.project | Default project | en_US |
| rioxxterms.version | NA | en_US |
| rioxxterms.versionofrecord | 10.7861/clinmed.22-4-s24 | en_US |
| rioxxterms.type | Journal Article/Review | en_US |
| refterms.dateFOA | 2022-11-01T14:00:57Z | |
| refterms.panel | Unspecified | en_US |
| html.description.abstract | Phosphate is important for normal mineralisation of bone. Phosphate is important in its own right for neuromuscular function, and profound hypophosphataemia can be accompanied by encephalopathy, muscle weakness and cardiomyopathy. Hypophosphataemia can be due to intracellular uptake of phosphate from the extracellular fluid, reduced intestinal phosphate absorption, increased renal excretion, decreased renal tubular absorptive capacity and genetic defects in renal tubule phosphate transporters. | en_US |
| rioxxterms.funder.project | 94a427429a5bcfef7dd04c33360d80cd | en_US |
